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Growth hormone release is closely coupled to slow-wave sleep, and the broader GHRH literature demonstrates that GHRH signaling can influence sleep-endocrine activity, particularly non-REM and slow-wave sleep parameters in controlled experimental settings [5]
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Although the detection of fatty acid content is not consistent across different tumors, these findings collectively suggest that PUFAs, especially arachidonic acid, are important inducers of lipid peroxidation in the TME
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